Benzos aren't used in cases wherein the ER staff suspect the patient is at risk of respiratory depression. Ketamine is effective in these cases, that's the point I was making, I could rephrase it though.
True! There's other posts in this vein on the blog. Most recent posts have concerned the NMDA Receptor, i'm mainly using ketamine to tease out intracellular effects of different NMDAR currents.
>At higher doses it start effecting other receptors which is why a higher does has seemingly opposite effects.
Also true, but not sure if that necessarily conflicts with what I'm writing about here in comparing Ketamine to Memantine.
>He does not seem to know ketamine is a D2 receptor agonsit, the same as PCP, yet he mentions it for PCP!
Learned this halfway through writing, forgot to amend that section. Completely on me.
Benzos aren't used in cases wherein the ER staff suspect the patient is at risk of respiratory depression. Ketamine is effective in these cases, that's the point I was making, I could rephrase it though.