Caffeine content for coffee, tea, soda and more(mayoclinic.org)
mayoclinic.org
Caffeine content for coffee, tea, soda and more
https://www.mayoclinic.org/healthy-lifestyle/nutrition-and-healthy-eating/in-depth/caffeine/art-20049372
21 comments
All black or green teas are not the same about caffeine, roasted black or green teas have less caffeine
Why wouldn’t they include a mg of caffeine per ounce (mL) column?
> Coffee drinks /Size in oz. (mL) /Caffeine (mg)
Brewed /8 (237) /96
Without the recipe, this table makes no sense.
Without the recipe, this table makes no sense.
What are some good decaf teas people enjoy?
Rooibus. It’s not decaf, it just has no caffeine. I drink it all the time since giving up coffee.
Rooibos is not tea in the strict sense of the word, it’s a completely different plant with a different set of bioactive alkaloids.
”The MeOH extract exhibited significant inhibition of the major human CYP450 isozymes (CYP3A4, CYP1A2, CYP2D6, CYP2C9, and CYP2C19). The strongest inhibition was observed by the extract for CYP3A4 (IC50 1.7 ± 0.1 μg/mL) followed by CYP2C19 (IC50 4.0 ± 0.3 μg/mL). Among the tested phytochemicals, the most potent inhibitors were isovitexin on CYP3A4 (IC50 3.4 ± 0.2 μM), vitexin on CYP2C9 (IC50 8.0 ± 0.2 μM), and thermopsoside on CYP2C19 (IC50 9.5 ± 0.2 μM). The two major, structurally related compounds aspalathin and nothofagin exhibited a moderate pregnane-X receptor (PXR) activation, which was associated with increased mRNA expression of CYP3A4 and CYP1A2, respectively. These results indicate that a high intake of nutraceuticals containing rooibos extracts may pose a risk of herb–drug interactions when consumed concomitantly with clinical drugs that are substrates of CYP enzymes.”
https://pubs.acs.org/doi/abs/10.1021/acs.jafc.9b00846
”The MeOH extract exhibited significant inhibition of the major human CYP450 isozymes (CYP3A4, CYP1A2, CYP2D6, CYP2C9, and CYP2C19). The strongest inhibition was observed by the extract for CYP3A4 (IC50 1.7 ± 0.1 μg/mL) followed by CYP2C19 (IC50 4.0 ± 0.3 μg/mL). Among the tested phytochemicals, the most potent inhibitors were isovitexin on CYP3A4 (IC50 3.4 ± 0.2 μM), vitexin on CYP2C9 (IC50 8.0 ± 0.2 μM), and thermopsoside on CYP2C19 (IC50 9.5 ± 0.2 μM). The two major, structurally related compounds aspalathin and nothofagin exhibited a moderate pregnane-X receptor (PXR) activation, which was associated with increased mRNA expression of CYP3A4 and CYP1A2, respectively. These results indicate that a high intake of nutraceuticals containing rooibos extracts may pose a risk of herb–drug interactions when consumed concomitantly with clinical drugs that are substrates of CYP enzymes.”
https://pubs.acs.org/doi/abs/10.1021/acs.jafc.9b00846
I don't drink decaffeinated tea, but I do drink quite a lot of naturally caffeine-free herbal teas. My local supermarket stocks dozens of varieties. I rather like peppermint but also enjoy other infusions depending on my mood.
Apparently all herbal teas don't contain caffeine. https://teaperfectionist.com/herbal-tea-benefits/
I'm partial to Yorkshire Gold. Bog-standard British builder's tea, but I can't taste the difference between the decaf and the regular.
I buy decaf Earl Grey from my local tea store. I cannot tell the difference; it's just another kind of Earl grey.
The list is endless, camomille, redbush, ginger are my favovorite non-caffeine teas.
Ginger with chamomile and tumeric is where it's at.
Corn silk tea and chamomile, both have zero caffeine.
I like Barley tea as my go to caffeine free tea.
Good link, I can see that I typically consume 150-300mg daily (A bit on the high side).
Can we have another table for total content of random substances acting as monoamine oxidase A or B inhibitors for coffee, tea, soda and more?